文 献1）Iijima K, Sako M, Nozu K, Mori R, Tuchida N, Kamei K, et al. Rituximab for childhood-onset, complicated, frequently relapsing nephrotic syndrome or steroid-dependent nephrotic syndrome: a multicentre, double-blind, randomised, placebo-controlled trial. Lancet 2014; 384(9950): 1273-81.2）Ito S, Kamei K, Ogura M, Udagawa T, Fujinaga S, Saito M, et al. Survey of rituximab treatment for childhood-onset refractory nephrotic syndrome. Pediatr Nephrol 2013; 28(2): 257-64.5）Kamei K, Takahashi M, Fuyama M, Saida K, Machida H, Sato M, et al. Rituximab-associated agranulocytosis in children with refractory idiopathic nephrotic syndrome: case series and review of literature. Nephrol Dial Transplant 2015; 30(1): 91-6.7）Kim DH, Jung HD, Kim JG, Lee JJ, Yang DH, Park YH, et al. FCGR3A gene polymorphisms may correlate with response to frontline R-CHOP therapy for diffuse large B-cell lymphoma. Blood 2006; 108(8): 2720-5.9）Jiménez Morales A, Maldonado-Montoro M, Martínez 3）Datta SS, Reddy M, Basu S, Krishnan S. Blood group discrepancy-first sign of autoimmune hemolytic anemia after hematopoietic stem cell transplantation in a child. Indian J Hematol Blood Transfus 2016; 32(Suppl 1): 211-3.4）Cattaneo C, Spedini P, Casari S, Re A, Tucci A, Borlenghi E, et al. Delayed-onset peripheral blood cytopenia after rituximab: frequency and risk factor assessment in a consecutive series of 77 treatments. Leuk Lymphoma 2006; 47(6): 1013-7.6）Keane C, Nourse JP, Crooks P, Nguyen-Van D, Mutsando H, Mollee P, et al. Homozygous FCGR3A-158V alleles predispose to late onset neutropenia after CHOP-R for diffuse large B-cell lymphoma. Intern Med J 2012; 42(10): 1113-9.8）Alberici F, Smith RM, Fonseca M, Willcocks LC, Jones RB, Holle JU, et al. Association of a TNFSF13B (BAFF) regulatory region single nucleotide polymorphism with response to rituximab in antineutrophil cytoplasmic antibody-associated vasculitis. J Allergy Clin Immunol 2017; 139(5): 1684-7 e10.de la Plata JE, Pérez Ramírez C, Daddaoua A, Alarcón Payer C, et al. FCGR2A/FCGR3A gene polymorphisms and clinical variables as predictors of response to tocilizumab and rituximab in patients with rheumatoid arthritis. J Clin Pharmacol 2019; 59(4): 517-31.10）Saito T, Ikeda M, Mushiroda T, Ozeki T, Kondo K, Shimasaki A, et al. Pharmacogenomic study of clozapine-induced agranulocytosis/granulocytopenia in a Japanese population. Biol Psychiatry 2016; 80(8): 636-42.11）Hallberg P, Eriksson N, Ibañez L, Bondon-Guitton E, Kreutz R, Carvajal A, et al. Genetic variants associated with antithyroid drug-induced agranulocytosis: a genome-wide association study in a European population. Lancet Diabetes Endocrinol 2016; 4(6): 507-16.12）Koido M, Kawakami E, Fukumura J, Noguchi Y, Ohori M, Nio Y, et al. Polygenic architecture informs potential vulnerability to drug-induced liver injury. Nat Med 2020; 26(10): 1541-8.13）Natarajan P, Young R, Stitziel NO, Padmanabhan S, Baber U, Mehran R, et al. Polygenic risk score identifies subgroup with higher burden of atherosclerosis and greater relative benefit from statin therapy in the primary prevention setting. Circulation 2017; 135(22): 2091-101.14）Abecasis GR, Altshuler D, Auton A, Brooks LD, Durbin RM, Gibbs RA, et al. A map of human genome variation from population-scale sequencing. Nature 2010; 467(7319): 1061-73.15）Altshuler DM, Gibbs RA, Peltonen L, Altshuler DM, Gibbs RA, Peltonen L, et al. Integrating common and rare genetic variation in diverse human populations. Nature 2010; 467(7311): 52-8.16）Purcell S, Neale B, Todd-Brown K, Thomas L, Ferreira MA, Bender D, et al. PLINK: a tool set for whole-genome association and population-based linkage analyses. Am J Hum Genet 2007; 81(3): 559-75.17）Flanagan J, Horikoshi M, Terao C. Reference panel using whole genome sequencing data in Japanese population (manuscript in submission). 2021.18）Das S, Forer L, Schönherr S, Sidore C, Locke AE, 107
元のページ ../index.html#121